Pulmonary insufficiency is occasionally a life-threatening sequel to nonthoracic trauma. This program includes a presentation on the effects of pulmonary insufficiency and possible treatments if it occurs. Charts, graphs, slides, x-rays and presentation of clinical patients to demonstrate the pathology of the lung lesion are included. Clinical phases of post-traumatic pulmonary insufficiency are defined and the current concepts on etiology and effective therapy are reviewed. According to this presentation, pulmonary insufficiency resulting from pulmonary shunting plays a large role in the deaths of 30-50 percent of adults in spite of intensive care following an episode of trauma, hemorrhage, major operation, or shock. The syndrome of post-traumatic pulmonary insufficiency can be broken down into four phases. Phase one consists of injury, hypocarbia, and respiratory and metabolic alkalosis. Phase two consists of circulatory stabilization in which there are signs of early respiratory problems including hypoxia and early physiologic shunting. Phase three consists of advancing pulmonary insufficiency with increasing acidosis and increasing pulmonary shunting. Phase four consists of terminal hypoxia, hypercarbia and asystole. The pathology of the lungs and the resuscitative measures are discussed. Therapy varies according to etiology. Basically it involves resuscitation, the early use of diuretics, early mechanical ventilation, controlled positive pressure breathing with the use of chest tubes, steroid therapy, the use of heparin when definite documented intravascular coagulation is present and, experimentally, the use of the membrane oxygenator.
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